Ginsburg I.  The biology of leukocyte-mediated proteolysis. Journal of Leukocyte Biology.  1999;66 (6) :1057.

Amar D, Roistacher N, Zhang H, Baum MS, Ginsburg I, Steinberg JS. Signal-averaged P-wave duration does not predict atrial fibrillation after thoracic surgery. Anesthesiology.  1999;91 (1) :16-23.

Ginsburg I, Sadovnic M, Varani J, Tirosh O, Kohen R. Hemolysis of human red blood cells induced by the combination of diethyldithiocarbamate (DDC) and divalent metals: Modulation by anaerobiosis, certain antioxidants and oxidants. Free Radical Research.  1999;31 (2) :79-91.

Barasch D, Zipori O, Ringel I, Ginsburg I, Samuni A, Katzhendler J. Novel anthraquinone derivatives with redox-active functional groups capable of producing free radicals by metabolism: are free radicals essential for cytotoxicity?. European Journal of Medicinal Chemistry.  1999;34 (7-8) :597-615.

Steinberg D, Heling I, Daniel I, Ginsburg I. Antibacterial synergistic effect of chlorhexidine and hydrogen peroxide against Streptococcus sobrinus, Streptococcus faecalis and Staphylococcus aureus. Journal of Oral Rehabilitation.  1999;26 (2) :151-156.

Ginsburg I, Sadovnik M, Sallon S, Milo-Goldzweig I, Mechoulam R, Breuer A, Gibbs D, Varani J, Roberts S, Cleator E, et al. PADMA-28, a traditional tibetan herbal preparation inhibits the respiratory burst in human neutrophils, the killing of epithelial cells by mixtures of oxidants and pro-inflammatory agonists and peroxidation of lipids. Inflammopharmacology.  1999;7 (1) :47-62.

Ginsburg I. Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae. Inflammopharmacology.  1999;7 (3) :207-217.

Ginsburg I. Is streptolysin S of group A streptococci a virulence factor?. APMIS.  1999;107 (12) :1051-1059.

Ginsburg I. "Cross-talk" among a multiplicity of pro-inflammatory agents: main cause of tissue damage in pulmonary inflammation?. European Respiratory Journal.  1999;14 (2) :481-482.

Ginsburg I, Ward PA, Varani J. Can we learn from the pathogenetic strategies of group A hemolytic streptococci how tissues are injured and organs fail in post-infectious and inflammatory sequelae?. FEMS Immunology & Medical Microbiology.  1999;25 (4) :325-338.